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Talking to Your Doctor About Cannabis: Bringing Up CBD and CBG

Effective healthcare hinges on precise communication. Telling your provider you 'use cannabis' is far too vague for a modern clinical environment. To ensure your treatment plan remains safe, provide specific data regarding the cannabinoids and exact dosages you use. A clear picture of your therapeutic goals helps your provider manage potential side effects or adverse drug interactions.

By Harrison

Why Precision Matters in the Exam Room

Vague descriptions of cannabis use hinder a provider's ability to offer accurate care. Specificity allows your provider to distinguish between casual use and a targeted wellness regimen. When you identify the exact compounds you consume, you help your provider monitor your physiology more effectively. Shifting from high-THC products to minor cannabinoids like CBG signals a calculated change in your symptom management strategy.

Distinguishing CBD from CBG

Understanding the functional differences between cannabinoids helps you articulate your self-titration process to your clinical team. These two compounds interact with your body in distinct ways.

How CBD (Cannabidiol) Functions

CBD is a non-intoxicating compound used by many for its calming properties. It works by inhibiting the FAAH enzyme, which may increase your body’s levels of anandamide—often called the "bliss molecule."

  • Common Uses: Patients use CBD for concerns related to generalized anxiety, systemic inflammation, and seizure management.
  • The Sensation: Users often describe the effect as a physical sense of relief without impairment to mental clarity.

How CBG (Cannabigerol) Integrates into Wellness

CBG is known as the "mother cannabinoid" because it is the chemical precursor to both THC and CBD. Unlike CBD, which acts indirectly, CBG binds directly to CB1 and CB2 receptors.

  • Common Uses: Research suggests CBG may support those managing Inflammatory Bowel Disease (IBD), symptoms of glaucoma, and bladder dysfunction.
  • The Sensation: Users often report a focused, "bright" feeling that supports daytime productivity, as it lacks the sedative qualities associated with other cannabis products.
Feature CBD (Cannabidiol) CBG (Cannabigerol)
Primary Interaction FAAH Enzyme / Serotonin CB1 & CB2 Receptors
Best For Anxiety, Sleep, Seizures Gut Health, Glaucoma, Focus
Liver Interaction High potential for drug interference Moderate potential
Common Effect Systemic Relaxation Targeted Anti-inflammatory

The Critical Link: Cannabis and Your Liver Enzymes

The most vital information you can share concerns the Cytochrome P450 (CYP450) enzyme system. This system in your liver processes approximately 60% of all prescribed medications.

Both CBD and THC utilize these same pathways. High doses of CBD can saturate these enzymes, potentially slowing the metabolic process for other drugs. Medications like Warfarin (blood thinners), Amiodarone (heart medication), or certain SSRIs may reach different levels in your bloodstream if your liver is preoccupied with metabolizing cannabinoids.

Suggested Script for Your Provider:

"I am taking 50mg of CBD oil daily. I want to ensure this isn't inhibiting my CYP3A4 or CYP2C19 enzymes. Can we monitor my blood levels to ensure my other medications are metabolizing correctly?"

Terpenes and Targeted Relief

Terpenes are the aromatic compounds that may influence the direction of a cannabis product's effect. Mentioning specific terpenes shows you are using cannabis with a clinical mindset.

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  • Myrcene: Associated with sedative properties; a choice for those struggling with insomnia or muscle spasms.
  • Beta-Caryophyllene (BCP): This unique terpene binds to CB2 receptors, providing anti-inflammatory benefits without causing drowsiness.

Using high-BCP products during the day allows you to manage pain while remaining alert.

Surgical Safety and Cannabis Disclosure

Honesty is a safety requirement before any surgical procedure. Cannabis use may impact how your body processes anesthesia.

  1. Anesthesia Resistance: Regular cannabis users may require higher doses of propofol to remain unconscious during surgery.
  2. Airway Management: Smoking or vaping can increase airway sensitivity, making intubation more complex for the surgical team.
  3. Heart Rate Fluctuations: Cannabis can contribute to tachycardia (rapid heart rate), which poses a risk to cardiovascular stability under general anesthesia.

Always inform your anesthesiologist of your last use and your typical daily dosage.

Building Your Cannabis Therapeutic Log

Data collection improves the quality of your consultations. Use a simple log to track your experiences and share the results with your provider.

Essential Data Points to Track:

  • Cannabinoid Ratios: Note the balance of CBD, CBG, and THC.
  • Delivery Method: Indicate if you are using tinctures, edibles, or vaporized flower.
  • Timing: Record when you dose in relation to your symptoms.
  • Side Effects: Document any instances of dry mouth, increased heart rate, or dizziness.

Managing the Provider-Patient Relationship

The goal of this disclosure is coordination of care. You are providing the data necessary for your provider to help keep you safe. If a provider dismisses your scientific approach or refuses to discuss the Endocannabinoid System (ECS), seek a provider who views your cannabis use as a meaningful variable in your overall health strategy. Professionals who understand cannabinoid medicine will appreciate your focus on safety and precision.


Legal Disclaimer: This content is for educational and informational purposes only and does not constitute medical advice. Always seek the advice of a physician regarding a medical condition. Efficacy has not been confirmed by FDA-approved research. Check your local laws regarding cannabis and terpene use.

Sources

  1. Blessing EM, Steenkamp MM, Manzanares J, Marmar CR. (2015). Cannabidiol as a potential treatment for anxiety disorders. Neurotherapeutics. 12(4):825-36. PubMed

  2. Russo EB. (2011). Taming THC: potential cannabis synergy and phytocannabinoid-terpenoid entourage effects. Br J Pharmacol. 163(7):1344-64. PubMed

  3. Borrelli F, Fasolino I, Romano B, Capasso R, Maiello F, Coppola D, Orlando P, Battista G, Clanning W, Di Marzo V, Izzo AA. (2013). Beneficial effect of the non-psychotropic plant cannabinoid cannabigerol on experimental inflammatory bowel disease. Biochem Pharmacol. 85(9):1306-16. PubMed

  4. Grotenhermen F, Müller-Vahl K. (2012). The therapeutic potential of cannabis and cannabinoids. Dtsch Arztebl Int. 109(29-30):495-501. PubMed

  5. VanDolah HJ, Bauer BA, Mauck KF. (2019). Clinicians' guide to cannabidiol and hemp oils. Mayo Clin Proc. 94(9):1840-51. PubMed

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